Compensatory Movements Observed in Hip Pain
Femoroacteabular impingement (FAI) is becoming more recognised as a mechanism in the development of hip pain and pathologies. Controlling hip and pelvic movements is a requirement for sport and actvities particulaly those involving high-flexion activities.
This paper has identified a compensatory posterior rotation of the pelvis on hip flexion in people with FAI. What comes first - we need to manage these uncontrolled movements but prevention is best and these and other movement faults need to be identifed in people at risk of developing FAI
The pelvifemoral rhythm in cam-type femoroacetabular impingement.
Van Houcke J, Pattyn C, Vanden Bossche L, Redant C, Maes JW, Audenaert EA.
Clinical Biomechanics 29 (2014) 63-67
There is growing evidence that femoroacetabular impingement is a potentially important risk factor for the development of early idiopathic osteoarthritis in the nondysplastic hip. Understanding of affected joint kinematics is a basic prerequisite in the evaluation of mechanical disorders in a clinical and research oriented setting. The aim of the present study was to compare pelvifemoral kinematics between subjects diagnosed with femoroacetabular impingement and healthy controls.
The authors collected motion data of the femur and pelvis on a total of 43 hips - 19 cam impingement hips and 24 healthy controls - using a validated electromagnetic tracking device. The pelvifemoral rhythm in supine position was defined during both active and passive hip flexion and statistically compared between both groups.
A significant increase in posterior pelvic rotation was observed during active hip flexion in the femoroacetabular impingement group compared with the control group (P<0.001). During passive hip flexion, however, posterior pelvic rotation between the impingement group and the controls did not differ significantly (P=0.628).
Posterior pelvic rotation during active high-end hip flexion is increased in femoroacetabular impingement, indicating the presence of an active compensational mechanism that decreases the extent of harmful joint conflict during high-flexion activities.
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